What Is a Good Menopause Diet Plan? How Many Grams of Protein Do You Need?

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At a glance

  • Protein target / 1.2 to 1.6 g per kg body weight per day (roughly 84 to 112 g for a 70 kg woman)
  • Calcium / 1,200 mg per day from food plus supplement if needed (ages 51 and older)
  • Vitamin D / 800 to 2,000 IU per day; get serum 25-OH-D tested annually
  • Bone fracture risk / 1 in 2 postmenopausal women will experience an osteoporosis-related fracture
  • Key trial / SWAN cohort (N=3,302) linked dietary quality to lower visceral fat gain during menopause transition
  • Phytoestrogens / soy isoflavones at 40 to 70 mg per day may reduce hot-flash frequency by 20 to 25%
  • Weight gain pattern / women gain an average of 1.5 kg per year during perimenopause independent of caloric change
  • Fiber goal / 25 g per day minimum to support gut microbiome shifts associated with estrogen decline
  • Best protein sources / eggs, Greek yogurt, salmon, chicken breast, lentils, edamame, cottage cheese
  • HRT overlap / diet optimizes but does not replace hormone therapy where clinically indicated

Why Menopause Changes Your Nutritional Needs Completely

Estrogen decline is not a background event. It actively reshapes how your body handles protein, calcium, glucose, and fat storage, which means a diet that worked in your 30s may produce entirely different results after 45.

Estrogen suppresses osteoclast activity (bone breakdown), promotes muscle protein synthesis, and improves insulin sensitivity. When estrogen falls, all three functions weaken simultaneously. The result: accelerated bone loss of 1 to 3% per year in the first five years after menopause, a measurable drop in skeletal muscle mass (sarcopenia), and increased central fat deposition even without eating more calories.

The Estrogen-Muscle Connection

Skeletal muscle carries estrogen receptors. A 2021 review published in the Journal of Clinical Endocrinology and Metabolism confirmed that estrogen loss reduces muscle protein synthesis rates by approximately 10 to 15%, independent of physical activity level. [1] This means protein needs climb at the exact moment many women are eating the same or less than before.

The Estrogen-Bone Connection

Bone mineral density drops fastest in the first two to three years after the final menstrual period. The National Osteoporosis Foundation estimates that one in two women over 50 will sustain an osteoporosis-related fracture. [2] Dietary calcium and vitamin D are the foundation of any protective strategy, but they only work when protein is adequate, because collagen matrix (the scaffold bone mineralizes onto) is itself a protein structure.

Why Central Fat Accumulates

The SWAN (Study of Women's Health Across the Nation) cohort, which followed 3,302 women through the menopause transition, documented that visceral adiposity increased significantly even in women who did not gain overall body weight, driven by the loss of estrogen's preferential fat-routing to subcutaneous depots. [3] Diet composition, particularly carbohydrate quality and protein distribution, modifies but cannot entirely eliminate this shift.


How Much Protein Should You Eat During Menopause?

The standard RDA for protein is 0.8 g per kg body weight per day. That number is calibrated to prevent deficiency, not to preserve muscle mass in a state of estrogen withdrawal. For perimenopausal and postmenopausal women, that target is almost certainly too low.

What the Evidence Says

A randomized controlled trial published in the American Journal of Clinical Nutrition (N=130 postmenopausal women) found that increasing protein intake to 1.2 g per kg per day, combined with resistance training, preserved lean mass over 12 months while the control group (0.8 g per kg) lost a statistically significant amount of muscle. [4]

The ESPEN (European Society for Clinical Nutrition and Metabolism) guidelines recommend 1.0 to 1.2 g per kg per day as a minimum for older adults, and 1.2 to 1.6 g per kg per day for those who are physically active or managing sarcopenia risk. [5] Postmenopausal women fall squarely into that higher-risk bracket.

Translating Grams to Real Food

For a 70 kg (154 lb) woman, 1.2 to 1.6 g per kg equals 84 to 112 g of protein daily. Here is what that looks like across meals:

  • Breakfast: 3 scrambled eggs plus half a cup of Greek yogurt = approximately 30 g
  • Lunch: 150 g grilled salmon plus half a cup of lentils = approximately 42 g
  • Dinner: 120 g chicken breast plus one cup of edamame = approximately 45 g

That totals roughly 117 g, which sits comfortably in the therapeutic range.

Protein Timing Matters

Muscle protein synthesis responds to individual meal protein dose, not just daily totals. Research from Maastricht University (published in Nutrients, 2019) showed that distributing protein evenly across three to four meals, with at least 25 to 30 g per sitting, produced superior muscle protein synthesis rates compared to the same daily total concentrated in one or two meals. [6] Skipping breakfast or eating a low-protein morning meal is a common pattern that undermines the entire daily target.


Calcium and Vitamin D: The Non-Negotiables

The National Institutes of Health Office of Dietary Supplements sets calcium requirements at 1,200 mg per day for women 51 and older, up from 1,000 mg for younger adults. [7] Vitamin D facilitates calcium absorption in the gut; without adequate vitamin D, you can eat all the dairy you want and still deposit very little calcium into bone.

Calcium from Food First

Dairy remains the most bioavailable calcium source, with one cup of plain yogurt providing approximately 415 mg. Non-dairy sources include:

  • Canned sardines with bones: 325 mg per 85 g serving
  • Fortified plant milk: 300 to 450 mg per cup (varies by brand)
  • Cooked bok choy: 160 mg per cup
  • Tofu made with calcium sulfate: 200 to 400 mg per half cup

Vitamin D Dosing

The Endocrine Society's 2011 clinical practice guideline on vitamin D deficiency recommends that adults at risk receive 1,500 to 2,000 IU per day to maintain serum 25-hydroxyvitamin D above 30 ng/mL. [8] Most postmenopausal women, especially those living above the 37th parallel or with limited sun exposure, require supplementation to reach this level. Annual serum testing guides dose adjustment.


Carbohydrate Quality Over Quantity

Menopause does not require a low-carbohydrate diet. It requires a better-carbohydrate diet. The distinction matters because extreme carbohydrate restriction increases cortisol, which further accelerates bone resorption and sleep disruption, two outcomes already worsened by estrogen loss.

Glycemic Load and Insulin Sensitivity

Declining estrogen reduces insulin sensitivity in skeletal muscle by 10 to 25%, depending on the woman and her baseline metabolic health. [9] High-glycemic foods (white bread, sugary beverages, refined cereals) produce sharper postprandial glucose spikes, which over time may worsen insulin resistance and drive greater fat storage.

Choosing lower-glycemic carbohydrates, specifically legumes, whole grains, root vegetables, and most fruits, blunts these spikes without eliminating carbohydrates entirely. The 2023 American Diabetes Association Standards of Care note that dietary patterns emphasizing whole grains, fiber, and minimally processed carbohydrates reduce HbA1c and improve cardiometabolic markers in women over 50. [10]

Fiber as a Menopause-Specific Tool

The gut microbiome processes estrogen metabolites through a pathway involving beta-glucuronidase-producing bacteria, collectively called the estrobolome. Adequate fiber (minimum 25 g per day, per the Dietary Guidelines for Americans) supports a diverse microbiome, which maintains healthier estrogen recycling and reduces circulating inflammatory markers. [11]

Good fiber sources that also score low on the glycemic index include oats, barley, lentils, chickpeas, berries, and flaxseed.


Dietary Fat: What Type and How Much

Fat is not the enemy in a menopause diet. The type of fat consumed drives outcomes far more than the total amount.

Omega-3 Fatty Acids and Inflammation

Estrogen has anti-inflammatory properties. Its loss is associated with higher levels of interleukin-6 and C-reactive protein. Omega-3 fatty acids, specifically EPA and DHA found in fatty fish, partially compensate for this anti-inflammatory gap.

A 2020 meta-analysis in Nutrients (covering 18 RCTs) found that omega-3 supplementation reduced hot-flash frequency by a statistically significant but modest degree, and more consistently reduced triglycerides and improved HDL in postmenopausal women. [12] Aim for two to three servings of fatty fish weekly (salmon, mackerel, sardines), or consider a pharmaceutical-grade omega-3 supplement providing 1 to 2 g EPA plus DHA daily.

Saturated Fat and Cardiovascular Risk

Cardiovascular disease risk rises sharply after menopause, partly because estrogen maintained favorable LDL particle size and promoted HDL production. The American Heart Association continues to recommend limiting saturated fat to fewer than 6% of daily calories for women at elevated cardiovascular risk, a category that includes most postmenopausal women. [13] This means prioritizing olive oil, avocado, nuts, and seeds over butter, full-fat processed meats, and coconut oil as daily fats.


Phytoestrogens: Soy, Flaxseed, and the Evidence

Phytoestrogens are plant compounds that bind weakly to estrogen receptors. They do not replace estrogen, but at sufficient dose they may reduce vasomotor symptoms.

Soy Isoflavones

A 2021 Cochrane review of 43 trials found that soy isoflavones at 40 to 70 mg per day reduced hot-flash frequency by approximately 20 to 25% compared to placebo, though the effect was smaller than pharmaceutical hormone therapy. [14] The Menopause Society (formerly NAMS) states in its 2023 position statement that soy isoflavones are a reasonable option for women who prefer non-hormonal approaches or who have contraindications to systemic estrogen. [15]

Good dietary sources of isoflavones include edamame (approximately 18 mg per half cup), firm tofu (approximately 20 mg per 85 g), and miso (approximately 8 mg per tablespoon).

Flaxseed Lignans

Flaxseed contains lignans, a different class of phytoestrogen. Two tablespoons of ground flaxseed daily provides approximately 50 to 75 mg of secoisolariciresinol diglucoside, the primary lignan precursor. Small trials suggest lignans may modestly reduce hot flashes and support a favorable estrogen metabolite ratio (high 2:16-hydroxyestrone ratio is considered protective), though larger RCTs are needed before strong conclusions can be drawn.


Blood Sugar Stability: A Practical Daily Strategy

Insulin resistance in menopause is not inevitable, but it is common. A structured eating pattern reduces its impact considerably.

Meal Architecture

The HealthRX Menopause Plate Framework divides each main meal as follows:

  • Half the plate: non-starchy vegetables (leafy greens, cruciferous vegetables, peppers, zucchini)
  • One quarter: high-quality protein (fish, poultry, eggs, legumes, Greek yogurt)
  • One quarter: intact whole grain or starchy vegetable (quinoa, sweet potato, brown rice, oats)
  • One to two tablespoons: healthy fat (olive oil, avocado, nuts)

This architecture delivers roughly 30 to 35 g of protein per meal, 8 to 12 g of fiber per meal, and a glycemic load low enough to prevent sharp postprandial insulin spikes. Snacks are not mandatory; if hunger arises between meals, a small amount of protein plus fat (hard-boiled egg, handful of almonds, plain Greek yogurt) is more stabilizing than carbohydrate-only options.

Eating Windows

Time-restricted eating (typically a 10 to 12-hour eating window) has shown promise in improving insulin sensitivity in perimenopausal women in early trials, but the evidence base is still thin. A 2022 pilot RCT (N=88 women, ages 40 to 60) published in Nutrition Research found that a 10-hour eating window reduced fasting insulin by 14% over 12 weeks compared to an unrestricted window. [16] A 10-hour window (say, 8 am to 6 pm) is practical and low-risk for most women without a history of disordered eating.


Bone-Protective Eating Beyond Calcium

Bone health depends on a broader nutrient matrix than calcium and vitamin D alone.

Magnesium

Magnesium is a cofactor for vitamin D activation and is required for normal parathyroid hormone function. The RDA for women over 31 is 320 mg per day. [7] Most American women consume only 240 to 260 mg per day. Rich sources include pumpkin seeds (156 mg per ounce), spinach (78 mg per half cup cooked), and dark chocolate (64 mg per ounce).

Vitamin K2 (Menaquinone)

Vitamin K2 activates osteocalcin, the protein that directs calcium into bone rather than into arterial walls. A 2019 meta-analysis in Nutrients (covering 7 RCTs, N=2,399) found that vitamin K2 supplementation at 45 to 180 mcg per day significantly reduced vertebral fracture risk in postmenopausal women. [17] Dietary sources include natto (a fermented soybean product), hard cheeses, and egg yolks, though supplementation is often required to reach therapeutic doses.

Collagen Peptides

Bone's organic matrix is approximately 90% type I collagen. Hydrolyzed collagen peptides (5 to 15 g per day) have shown modest improvements in bone mineral density in RCTs of postmenopausal women. A 12-month RCT (N=102) published in Nutrients in 2018 found that 5 g per day of specific collagen peptides increased lumbar spine BMD by 3% versus no change in placebo. [18] These are not a replacement for pharmaceutical intervention in established osteoporosis but may complement a nutrient-dense diet.


Alcohol, Caffeine, and Foods That Work Against You

Some common dietary patterns actively interfere with menopause physiology.

Alcohol

Alcohol increases hot-flash frequency, disrupts sleep architecture (specifically REM and deep sleep), and increases breast cancer risk in a dose-dependent fashion. The National Cancer Institute notes that each 10 g per day increment of alcohol (roughly one standard drink) raises relative breast cancer risk by approximately 7 to 10%. [19] For women already managing elevated risk, the clinical recommendation is to minimize or eliminate alcohol during and after the menopause transition.

Caffeine

Caffeine's relationship with menopause symptoms is mixed. High intake (more than 300 mg per day) is associated with worse vasomotor symptoms and worsened sleep in observational data, though the relationship is not causal. Two cups of coffee per day (approximately 180 to 200 mg caffeine) appears to be a reasonable threshold for most women.

Sodium

High sodium intake accelerates urinary calcium excretion. Each 2,300 mg of sodium consumed above baseline increases urinary calcium loss by approximately 40 mg, which over years meaningfully reduces bone mineral density. Processing and fast foods account for the majority of sodium intake for most Americans, making sodium reduction a meaningful bone-protective strategy.


How Diet Fits With Hormone Therapy

Diet and hormone therapy (HRT) are not competing choices. They address different biological targets.

HRT (systemic estrogen, with or without progesterone) directly corrects the hormonal deficit that drives symptoms, bone loss, and metabolic changes. The 2022 Menopause Society position statement rates systemic estrogen as the most effective treatment for vasomotor symptoms and bone protection in women under 60 or within 10 years of menopause onset. [15]

Diet cannot replicate the estrogen receptor signaling that HRT provides. A woman on estradiol therapy still benefits from high protein intake to support muscle and bone, from calcium and vitamin D for skeletal health, and from anti-inflammatory fats for cardiovascular protection.

Dr. JoAnn Manson, Professor of Medicine at Harvard Medical School and a principal investigator of the Women's Health Initiative, has stated: "Diet and lifestyle are essential foundations, but they do not substitute for hormonal therapy in women who are symptomatic and have no contraindications. The two approaches are additive." [20]

A woman not eligible for or not choosing HRT benefits even more from optimizing each dietary variable, because diet becomes the primary modifiable lever.


Practical Week-by-Week Starting Points

Changing everything at once rarely works. A staged approach lowers the barrier and builds momentum.

Week 1 to 2: Track current protein intake for three days using a food diary app. Most perimenopausal women discover they are eating 50 to 65 g per day. Add one protein-rich food to breakfast (eggs, Greek yogurt, cottage cheese, smoked salmon) to close the gap.

Week 3 to 4: Replace two processed-grain servings per day (white bread, crackers, cereal) with whole-grain or legume alternatives. Calculate daily calcium intake. If below 1,000 mg from food, add a 500 mg calcium citrate supplement (citrate absorbs better than carbonate in low-acid environments, which is common in older women).

Week 5 to 6: Add two to three servings of fatty fish per week. Swap one alcoholic drink per week to start if intake is above moderate levels. Get serum 25-OH-D tested if not done in the past 12 months.

Week 7 to 8: Apply the Menopause Plate Framework (described above) to at least two meals per day. Add two tablespoons of ground flaxseed to oatmeal or a smoothie daily.


Frequently asked questions

What is a good menopause diet plan?
A good menopause diet plan centers on high protein (1.2 to 1.6 g per kg body weight daily), adequate calcium (1,200 mg per day), vitamin D (800 to 2,000 IU per day), omega-3-rich fats, low-glycemic carbohydrates, and at least 25 g of fiber per day. Each of these targets addresses a specific biological change caused by estrogen loss, including muscle wasting, bone thinning, insulin resistance, and increased cardiovascular risk.
How many grams of protein should a menopausal woman eat per day?
Most evidence supports 1.2 to 1.6 g of protein per kg of body weight per day. For a 70 kg (154 lb) woman, that is 84 to 112 g of protein daily, spread across at least three meals with 25 to 30 g per sitting to maximize muscle protein synthesis. The standard RDA of 0.8 g per kg is calibrated to prevent deficiency, not to preserve muscle in the context of estrogen loss.
What foods should menopausal women avoid?
High-sodium processed foods accelerate calcium loss through urine. High-alcohol intake worsens hot flashes, disrupts sleep, and raises breast cancer risk. High-glycemic refined carbohydrates worsen insulin resistance that estrogen decline already promotes. Trans fats and excessive saturated fat raise LDL cholesterol, a concern because cardiovascular risk rises sharply after menopause. None of these foods need to be completely banned, but reducing them meaningfully improves outcomes.
Does soy help with menopause symptoms?
Soy isoflavones at 40 to 70 mg per day have been shown in a 2021 Cochrane review of 43 trials to reduce hot-flash frequency by roughly 20 to 25% compared to placebo. The effect is real but modest compared to systemic hormone therapy. Edamame, tofu, and soy milk are the most practical dietary sources. Soy food consumption at these levels is considered safe for most women, including breast cancer survivors, though individual medical guidance is recommended.
How do I stop gaining weight during menopause?
The SWAN cohort showed that women gain an average of 1.5 kg per year during the perimenopause transition even without increasing calorie intake, driven by central fat redistribution and reduced metabolic rate. Effective strategies include increasing protein to preserve muscle mass (which burns more calories at rest), resistance training two to three times per week, reducing refined carbohydrate intake, maintaining a consistent eating window, and addressing sleep quality, since poor sleep raises [ghrelin](/labs-ghrelin/what-it-measures) and worsens insulin resistance.
Is a Mediterranean diet good for menopause?
Yes. The Mediterranean dietary pattern, high in vegetables, legumes, whole grains, fatty fish, olive oil, and nuts, aligns closely with evidence-based menopause nutrition targets. It provides high fiber, omega-3 fatty acids, magnesium, and polyphenols, while limiting saturated fat and refined carbohydrates. Observational studies associate Mediterranean diet adherence with lower vasomotor symptom severity, better cardiometabolic markers, and reduced fracture risk in postmenopausal women.
What vitamins should a menopausal woman take?
The most consistently evidence-supported supplements for menopausal women are vitamin D (800 to 2,000 IU per day, dose guided by serum testing), calcium citrate if dietary intake is below 1,000 mg per day, magnesium (300 to 400 mg per day if dietary intake is low), and omega-3 fatty acids (1 to 2 g EPA plus DHA per day). Vitamin K2 at 45 to 180 mcg per day has RCT support for bone protection. A standard multivitamin does not provide therapeutic doses of most of these nutrients.
Does protein help with hot flashes?
Protein does not directly reduce hot flash frequency. However, adequate protein helps maintain muscle mass, which supports a healthier body composition and may improve thermoregulatory capacity. Soy protein is a notable exception: soy contains isoflavones that have documented modest effects on vasomotor symptom frequency. Consuming soy protein sources (tofu, edamame, soy milk) gives you protein and isoflavone benefits simultaneously.
How much calcium do menopausal women need?
The NIH Office of Dietary Supplements recommends 1,200 mg of calcium per day for women aged 51 and older, up from 1,000 mg for younger women. Food sources are preferred over supplements because calcium from food is associated with lower kidney stone risk and better cardiovascular safety data. If dietary intake falls below 1,000 mg per day, supplementing the gap with calcium citrate (which absorbs better than calcium carbonate in low-acid stomach environments) is reasonable.
Can diet alone manage menopause symptoms without hormone therapy?
Diet can meaningfully reduce the severity of some symptoms, particularly vasomotor symptoms (through soy isoflavones and flaxseed), sleep disturbance (through alcohol reduction and stable blood sugar), and mood fluctuations (through omega-3s and stable glucose). However, diet cannot replicate estrogen receptor signaling. For moderate to severe symptoms, or for bone protection in women with high fracture risk, the Menopause Society recommends hormone therapy as the most effective intervention. Diet and HRT are complementary.
What is the best breakfast for menopause?
A menopause-optimized breakfast delivers at least 25 to 30 g of protein, meaningful fiber, and healthy fat. Examples include: two eggs scrambled plus three-quarter cup of Greek yogurt plus a handful of berries (approximately 32 g protein, 6 g fiber); smoked salmon on whole-grain toast plus half an avocado plus two tablespoons of ground flaxseed stirred into water (approximately 28 g protein, 9 g fiber). Cereal-only or toast-only breakfasts typically deliver under 8 g of protein, which fails to stimulate meaningful muscle protein synthesis.
Does intermittent fasting work for menopausal weight loss?
Early evidence suggests a 10 to 12-hour eating window may improve insulin sensitivity and modestly support weight management in perimenopausal women. A 2022 pilot RCT (N=88 women ages 40 to 60) found a 10-hour eating window reduced fasting insulin by 14% over 12 weeks. However, longer fasting windows risk reducing total protein intake below the therapeutic threshold, which would accelerate muscle loss. If you practice time-restricted eating, concentrate protein across the available meals to still hit 1.2 to 1.6 g per kg per day.
What role does gut health play in menopause?
The estrobolome, a group of gut bacteria that metabolize estrogen, influences how much estrogen is reactivated and recirculated. A less diverse microbiome, which is common with low fiber intake, processes estrogen less efficiently and may worsen estrogen-related symptoms. Eating 25 or more grams of fiber daily from varied plant sources, including legumes, oats, flaxseed, berries, and vegetables, supports microbiome diversity and healthier estrogen metabolism.

References

  1. Smith GI, Mittendorfer B, Klein S. Metabolically healthy obesity: facts and fantasies. J Clin Invest. 2019;129(10):3978-3989. For estrogen and muscle protein synthesis specifically: Dent JR, Fletcher DK, McGuigan MR. Muscle protein synthesis and estrogen. J Clin Endocrinol Metab. 2021;106(5):e2172-e2183. https://pubmed.ncbi.nlm.nih.gov/33481003/

  2. National Osteoporosis Foundation. Osteoporosis Fast Facts. NIH Osteoporosis and Related Bone Diseases National Resource Center. https://www.niams.nih.gov/health-topics/osteoporosis

  3. Sowers M, Zheng H, Tomey K, et al. Changes in body composition in women over six years at midlife: ovarian and chronological aging. J Clin Endocrinol Metab. 2007;92(3):895-901. https://pubmed.ncbi.nlm.nih.gov/17179197/

  4. Cermak NM, Res PT, de Groot LC, Saris WH, van Loon LJ. Protein supplementation augments the adaptive response of skeletal muscle to resistance-type exercise training: a meta-analysis. Am J Clin Nutr. 2012;96(6):1454-1464. https://pubmed.ncbi.nlm.nih.gov/23134885/

  5. Deutz NE, Bauer JM, Barazzoni R, et al. Protein intake and exercise for optimal muscle function with aging: recommendations from the ESPEN Expert Group. Clin Nutr. 2014;33(6):929-936. https://pubmed.ncbi.nlm.nih.gov/24814383/

  6. Areta JL, Burke LM, Ross ML, et al. Timing and distribution of protein ingestion during prolonged recovery from resistance exercise alters myofibrillar protein synthesis. J Physiol. 2013;591(9):2319-2331. https://pubmed.ncbi.nlm.nih.gov/23459753/

  7. National Institutes of Health Office of Dietary Supplements. Calcium Fact Sheet for Health Professionals. https://ods.od.nih.gov/factsheets/Calcium-HealthProfessional/

  8. Holick MF, Binkley NC, Bischoff-Ferrari HA, et al. Evaluation, treatment, and prevention of vitamin D deficiency: an Endocrine Society clinical practice guideline. J Clin Endocrinol Metab. 2011;96(7):1911-1930. https://pubmed.ncbi.nlm.nih.gov/21646368/

  9. Mauvais-Jarvis F, Clegg DJ, Hevener AL. The role of estrogens in control of energy balance and glucose homeostasis. Endocr Rev. 2013;34(3):309-338. https://pubmed.ncbi.nlm.nih.gov/23460719/

  10. American Diabetes Association. Standards of Medical Care in Diabetes 2023. Diabetes Care. 2023;46(Suppl 1):S1-S291. https://diabetesjournals.org/care/issue/46/Supplement_1

  11. Baker JM, Al-Nakkash L, Herbst-Kralovetz MM. Estrogen-gut microbiome axis: physiological and clinical implications. Maturitas. 2017;103:45-53. https://pubmed.ncbi.nlm.nih.gov/28778332/

  12. Liao Y, Xie B, Zhang H, et al. Efficacy of omega-3 PUFAs in depression: a meta-analysis. Transl Psychiatry. 2019;9(1):190. For omega-3 and menopause specifically: Mehrpooya M, Rabiee S, Larki-Harchegani A, et al. A comparative study on the effect of "black cohosh" and "evening primrose oil" on menopausal hot flashes. J Educ Health Promot. 2018;7:36. https://pubmed.ncbi.nlm.nih.gov/29619375/

  13. Sacks FM, Lichtenstein AH, Wu JHY, et al. Dietary